The Anatomy Behind the Penis Enlargement Response
To understand the precise mechanism of the penis enlargement response, it’s important to first understand the anatomy of the penis. The penis is a specialized vascular framework constructed to inflate to a rigid state called — erection. The shaft consists of three chambers:
- The two chambers of the corpus spongiosum (CS) and
- The single chamber of the corpus cavernosum (CC).
Within these chambers are prominent slit like spaces, rich in blood cells, as they are really modified veins called vascular plexus. The slit like vascular spaces are composed of an abundant amount of smooth muscle with lesser amounts of fibrous tissue, lined by endothelium. This is called erectile tissue.
Separating the CC and CS is a very thick band of connective tissue called the tunica albuginea. This connective tissue is composed mostly of bi-layered collagen and elastic fibers. The elastic fibers form an irregularly latticed network on which collagen fibers rest. It is this elastic network that gives the penis elasticity, allowing it to expand and contract, and is an important factor in the penis enlargement response.
The Limiting Factor of the Penis Enlargement Response
The limiting factor of the penis enlargement response is that the composition of the tunica albuginea and septum is collagen and elastic tissue. The collagen is very tough connective tissue, constructed by nature, to ensure optimum structural integrity and strength. Think of a tendon or ligament. They’re also composed of collagen. Your body’s entire structural framework is composed mostly of collagen, including bone.
To facilitate the penis enlargement response, you must overcome the structural bonds of the collagen. Through sufficient traction, called critical traction, applied consistently along the length and diameter of the penis, the molecular bonds of the collagen will separate and undergo remodeling to a larger size.
Breaking the Bonds for the Penis Enlargement Response
The interior tissue of the penis is composed mainly of the vascular plexus, consisting of epithelium lining, with abundant smooth muscles and some fibrous tissue. When sufficient traction is applied to the collagen, this results in a controlled bond breaking and separation. The penis enlargement response and the remodeling process begins, making the collagen longer and larger.
Think of separating a brick wall and filling it in with additional bricks resulting in a larger brick wall. During the penis enlargement response, the interior vascular plexus goes along for the ride, but itself is transformed into greater mass.
The endothelium lining of the vascular slits undergoes mitosis, during the penis enlargement response. The abundant smooth muscle framework adjacent to the endothelium undergoes hypertrophy and mitosis (there are studies that support the proposition that mitosis can occur in smooth muscle cells), and the arteries and veins follow the same growth course mentioned above, with the remaining fibrous tissue undergoing remodeling.
The slow-growing, peripheral nerves grow and elongate when stimulated during the penis enlargement response. Therefore, the entire package gets bigger under sufficient constructive, critical traction, with the collagenous structure being the limiting factor. If the collagen bond breaking is sufficient, with cementing, the process is likely permanent.
Newbie Gains and the Penis Enlargement Response
Let’s take a brief look at “newbie gains” and the penis enlargement response.
A “newbie” is a person who has never tried penis enlargement. Once he begins, he will experience characteristically easy gains. Unchallenged or unstressed collagen has slack or crimps, allowing a stretching out scenario that facilitates the penis enlargement response.When these crimps are removed, it results in quick gains for the beginner. Under this condition, the collagen bonds generally remain intact until the critical traction level is applied.
Ligaments and the Penis Enlargement Response
Another factor in the penis enlargement response is the ligaments. The attachment points for the penis are composed of collagenous tissue, called ligaments, and skeletal muscular tissue. The main ligaments are called suspensory ligaments and fundiform ligaments. The primary base muscles are called the bulbocavernosus and ischiocavernosus muscles.
Ligaments are composed of densely-packed collagen fibers, which are white and relatively inelastic. Mechanical properties vary with shape and structural organization. This connective tissues is characterized by sparse cellularity, and distributed within an extracellular matrix.
Cells in tendons and ligaments are called fibroblasts. These ligaments consist of 70-80% collagen and 20% fibroblasts. Upon initiating traction, the crimps of the ligament collagen are pulled out or straightened first, as part of the penis enlargement response. After this, additional traction triggers molecular transformation via molecular bond breaking and remodeling, again resulting in longer, larger ligaments, as the penis enlargement response.
Skeletal Muscles and the Penis Enlargement Response
Another factor in the penis enlargement response is the skeletal muscles. The skeletal muscles called the bulbocavernosus and ischiocavernosus muscles, are attachment points at the base of the penis. For this reason, traction will elicit the same changes in the penis’s skeletal muscles as arm curls will elicit in your biceps and triceps. Whilst under traction, all muscular tissue at the base of the penis will undergo hypertrophy, strengthen and thus initiate the penis enlargement response.
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