Pre e is a physical cause, caused by rushed masturbation, light grip, lack of stimulation / pleasure. Anxiety doesn't help, like with anything,depression, weight gain etc. but isn't a cause.
Arousal also doesn't help cumming sooner than you want but again isn't a cause.
Chemical imbalance is bollox, if you post to disagree give some evidence or reasons why.
Reason anxiety is believed to be a cause is because there are meds for anxiety delay ejac. Ssri's increase serotonin which delays ejac, i've looked into it and can't find proof of a direct link between serotonin increase and delayed ejac. Serotonin regulates mood, appetite, memory, libido and aparrently sexual performance ( ejaculatory response ) but as i said nothing directly links the two.
Many ssri's have physical affects which seem to impact delay of orgasm more than increase in serotonin.
Tramadol is also a painkiller ( opiod ) and can have mild sedative affects.
Dxm ( couch meds ) has muscle relaxant affects.
Mdma, extacy also produces high levels of serotonin, but have numbing affects on the body ( aswel as mixed agents ). Same for cocaine which is a dopamine agonist.
This is the most interesting. Prozac, zoloft, paxil and celexa commonly cause Vasodilation (or*vasodilatation). This is the widening of*blood vessels. It results from relaxation of*smooth muscle*cells within the vessel walls, particularly in the large*veins, large*arteries, and smaller arterioles. The process is the opposite of*vasoconstriction, which is the narrowing of blood vessels. When blood vessels dilate, the flow of blood is increased due to a decrease in vascular resistance.Therefore, dilation of arterial blood vessels (mainly the arterioles) decreases*blood pressure. The response may be intrinsic (due to local processes in the surrounding tissue) or extrinsic (due to*hormones*or the*nervous system). In addition, the response may be localized to a specific organ (depending on the metabolic needs of a particular tissue, as during strenuous exercise), or it may be systemic (seen throughout the entiresystemic circulation).
Edit:
Several authors have reported their experience with PDE5 inhibitors alone or in combination with SSRIs as a treatment for prem e.
The proposed mechanisms for the effect of sildenafil (viagra ) on ejaculatory latencies include a central effect involving increased NO and reduced sympathetic tone, smooth muscle dilatation of the vas deferens and seminal vesicles, which oppose sympathetic vasoconstriction and delay ejaculation.
High blood pressure ( caused by vasoconstriction ) medication is also prescribed for premature ejaculation, though not as commonly.
Edit 2:
Looking into magnesium, I know that sameel posted up about histamine and magnesium deficiency being linked to prem e. So from researching, magnesium deficiencies increase the activity of vasoconstriction.
http://www.google.com.au/url?sa=t&rc...yvpXJxofCq20QA
Here is an article posted by Pegasus. There is mention of the use of ssri's and pde5 inhibitors increasing ielt ( time of ejaculation after insertion ) and how the pelvic floor muscle rehabilitation does the same.
Prove me wrong neuro bitches.
Arousal also doesn't help cumming sooner than you want but again isn't a cause.
Chemical imbalance is bollox, if you post to disagree give some evidence or reasons why.
Reason anxiety is believed to be a cause is because there are meds for anxiety delay ejac. Ssri's increase serotonin which delays ejac, i've looked into it and can't find proof of a direct link between serotonin increase and delayed ejac. Serotonin regulates mood, appetite, memory, libido and aparrently sexual performance ( ejaculatory response ) but as i said nothing directly links the two.
Many ssri's have physical affects which seem to impact delay of orgasm more than increase in serotonin.
Tramadol is also a painkiller ( opiod ) and can have mild sedative affects.
Dxm ( couch meds ) has muscle relaxant affects.
Mdma, extacy also produces high levels of serotonin, but have numbing affects on the body ( aswel as mixed agents ). Same for cocaine which is a dopamine agonist.
This is the most interesting. Prozac, zoloft, paxil and celexa commonly cause Vasodilation (or*vasodilatation). This is the widening of*blood vessels. It results from relaxation of*smooth muscle*cells within the vessel walls, particularly in the large*veins, large*arteries, and smaller arterioles. The process is the opposite of*vasoconstriction, which is the narrowing of blood vessels. When blood vessels dilate, the flow of blood is increased due to a decrease in vascular resistance.Therefore, dilation of arterial blood vessels (mainly the arterioles) decreases*blood pressure. The response may be intrinsic (due to local processes in the surrounding tissue) or extrinsic (due to*hormones*or the*nervous system). In addition, the response may be localized to a specific organ (depending on the metabolic needs of a particular tissue, as during strenuous exercise), or it may be systemic (seen throughout the entiresystemic circulation).
Edit:
Several authors have reported their experience with PDE5 inhibitors alone or in combination with SSRIs as a treatment for prem e.
The proposed mechanisms for the effect of sildenafil (viagra ) on ejaculatory latencies include a central effect involving increased NO and reduced sympathetic tone, smooth muscle dilatation of the vas deferens and seminal vesicles, which oppose sympathetic vasoconstriction and delay ejaculation.
High blood pressure ( caused by vasoconstriction ) medication is also prescribed for premature ejaculation, though not as commonly.
Edit 2:
Looking into magnesium, I know that sameel posted up about histamine and magnesium deficiency being linked to prem e. So from researching, magnesium deficiencies increase the activity of vasoconstriction.
http://www.google.com.au/url?sa=t&rc...yvpXJxofCq20QA
Here is an article posted by Pegasus. There is mention of the use of ssri's and pde5 inhibitors increasing ielt ( time of ejaculation after insertion ) and how the pelvic floor muscle rehabilitation does the same.
Prove me wrong neuro bitches.
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