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Pegasus · 05-05-2020, 08:20 PM

Going to post links for various stuff which may be of interest .
This has glute bridge in it .
https://www.pegym.com/forums/penile-...lvic-tilt.html

This is a compilation of stretches which may be useful some links may be broken keep going with it .

https://www.pegym.com/forums/prematu...ber-input.html

This is written during covid lockdown so for those who can't get to a physio check this
https://www.pegym.com/forums/penile-...=habs+protocol

Pegasus · 05-05-2020, 08:38 PM

https://e5v4d7a2.stackpathcdn.com/wp...160.jpg?x75358

Hard flacid discussion group.

Pegasus · 05-05-2020, 08:53 PM

I reproduce an article I wrote which was published onsite . Gives an overview of the cuting edge of mainstream thought on hf.

Hard Flaccid: Beyond The Edge Of Science?

By Pegasus
Pegasus acknowledges the assistance of Hans and Al in the creation of this article.
PEGym deals with issues of male sexuality covering a number of areas. A difficult, frustrating and somewhat mysterious issue has been Hard flaccid (HF). This covers a wide range of symptoms:
– Pelvic pain
– Pain on ejaculation
– Retracted penis
– A “rubbery” feeling to the penis
– Flaccid glans during an erection
– Changes in penis shape and size
– Loss of libido
– Difficulty getting an erection.
Over time, the mods onsite have recommended 2 main courses of action: stress reduction/dealing with stress anxiety, and on the physical side pelvic floor work to relax the pelvic floor. Our working theory was that on the physical level spasm /tension within the pelvic floor interfered with blood flow and nerve conduction. We also believed stress to be frequently causal in both the difficulty getting erect development and maintain of this syndrome.
Problem was we got a lot of blowback from members with HF on a number of fronts. The most common was the assertion that the problem was totally physical and stress was just a side effect and our view on mental states was demeaning and a distraction. However we also were told hey the problem is in my penis not my pelvis and the cure lies elsewhere. Now the view from actual suffers on what treatment should be was all over place the importance of the mental side was variously dismissed or regarded as central or indeed the only factor. On the physical side a wide range of approaches from NoFap/chastity through fasting through a wide range of physical therapies to just forgetting about it and getting on with life.
So I have been interested in the male pelvic floor and it’s effect on male sexuality for some time. It has been of great interest to me to see the development of mainstream medical views on this relationship in recent years. The development in male pelvic floor physio has been of particular interest. So early in the piece I went looking for the science on HF and found uuummm well nothing. However very recently hallelujah I see the start of discussion about what HF is and a move to recognise it. Some uros have begun the process to recognise it as a condition. More importantly some brave physio are going beyond the science to look at actual treatment.
From urology news:
Hard flaccid syndrome
By Kaylie Hughes, Arie Parnham and Marc Lucky
“However, a new phenomenon, known as hard flaccid syndrome, is being recognised as an alternative cause of unremitting pelvic pain in men by specialist pelvic floor physiotherapists and some urologists. Currently, no published literature exists of the syndrome”
Later they say:
“Hard flaccid syndrome is a type of CPPS.”
Then later:
“The true aetiology of this condition is unknown. Like many CPPS the development of hard flaccid syndrome is thought to be multifactorial in nature. Biological, psychological and social influences all contribute to the development and severity of the condition by altering the neurovascular supply to the muscles of the pelvic floor and penis.
“Stress is a key risk factor for the development of this condition by way of causing prolonged contraction of the muscles of the pelvic floor. Stress can be triggered by an injury directly to the penis during sexual intercourse or masturbation, or stress secondary to psychosocial distress in the absence of injury.”
Entropy Physio speculates it is a form of CPPS and has this to say:
“First things first: there is nothing wrong with your/your client’s penis. Often, clients have been to many providers and have been given clean bills of health He is shooed away with pharmaceuticals and no explanation why his friend is under the weather. Ruminating on the issue can precipitate a key ingredient to the problem: stress. Stress causes activation of your sympathetic nervous system which in turn releases adrenaline throughout your body.”
So they believe it is physically pelvic and that mental factors are central.
Urologist K .Hughes believes:
“Hard flaccid syndrome is difficult to treat due to the fact it is poorly understood and not yet widely recognised as a condition by urologists. Like with any CCPS, adoption of a multimodality holistic approach is paramount when managing these men. The most important step in the management of this condition is continued reassurance that physically there is nothing functionally wrong with the penis and that this is a chronic pain syndrome.”
The physio at Core Body Clinic’s view is:
“Hard Flaccid syndrome has largely been the subject of discussion and debate on male pelvic pain forums with little or no clinical information about the condition. Therefore, diagnosis is difficult to the untrained eye and often clinicians are often at a loss about treatment.”
Then later:
“With little by way of clinical research concerning hard flaccid, much of the theories relating to the pathophysiology are based on our observations of the patients who present in clinic. However, in combination with the knowledge of anatomy and pain mechanisms an understanding has been developed which has enabled us to provide treatment solutions.
“Injury to the penis and stress are likely drivers for this condition”
Leading physio Gerard Greene says in a podcast that HF is largely pelvic floor related at a physical level and also talks on the effect of stress . This from a patient of his:
“I had heard of Hard Flaccid before but always wondered if it really existed as I could never find anything on google about it but had seen it mentioned in one forum. Without me mentioning it Gerard thought I had pelvic pain definitely but also Hard flaccid. The way he described it , and I’m not a medical person , is that if there is pain for a long time in the pelvis and nerves get irritated or inflamed ( pudendal & others ) then the tissues those nerves supply can get affected i.e the penis.”
So it seems the mods at PEGym were on the right track. HF at the physical level is a pelvic floor issue and stress is heavily involved.
References:
Laumann EO, Nicolosi A, Glasser DB, Paik A, Gingell C, Moreira E, et al. Sexual problems among women and men aged 40-80 y: prevalence and correlates identified in the Global Study of Sexual Attitudes and Behaviors. Int J Impot Res. 2005;17:39–57.
Nickel JC, Freedland SJ, Castro-Santamaria R, Moreira DM. Chronic prostate inflammation predicts symptom progression in patients with chronic prostatitis/chronic pelvic pain. J Urol. 2017;198:122–8.
Labat JJ, Robert R, Delavierre D, et al. Anatomy and physiology of chronic pelvic and perineal pain. Prog Urol 2010;20(12):843-52.
Dybowski C, L�we B, Br�nahl C. Predictors of pain, urinary symptoms and quality of life in patients with chronic pelvic pain syndrome (CPPS): A prospective 12-month follow-up study. J Psychosom Res 2018;112:99-106.
Passavanti MB, Pota V, Sansone P, et al. Chronic pelvic pain: assessment, evaluation, and objectivation. Pain Res Treat 2017;9472925.
Sandhu J, Tu HYV. Recent advances in managing chronic prostatitis/chronic pelvic pain syndrome. F1000Res 2017;6.pii:F1000 Faculty Rev-1747.
Quaghebeur J, Wyndaele JJ. Prevalence of lower urinary tract symptoms and level of quality of life in men and women with chronic pelvic pain. Scand J Urol 2015;49(3):242-9.
Jansen AS, Nguyen XV, Karpitskiy V, et al. Central command neurons of the sympathetic nervous system: basis of the fight-or-flight response. Science 1995;270(5236):644-6.
Jantos M. Understanding chronic pelvic pain. Pelviperineology 2007;26:66-9.
Hubbard DR. Chronic and recurrent muscle pain: Pathophysiology and treatment, and review of pharmacologic studies. J Musculoskeletal Pain 1996;4:123-43.
Hannibal KE, Bishop MD. Chronic stress, cortisol dysfunction, and pain: a psychoneuroendocrine rationale for stress management in pain rehabilitation. Physical Therapy 2014;94(12):1816–25.
Krsmanovic A, Tripp DA, Nickel JC, et al. Psychosocial mechanisms of the pain and quality of life relationship for chronic prostatitis/chronic pelvic pain syndrome (CP/CPPS). Can Urol Assoc J 2014;8(11-12):403-8.
Montenegro ML, Gomide LB, Mateus-Vasconcelos EL, et al. Abdominal myofascial pain syndrome must be considered in the differential diagnosis of chronic pelvic pain. Eur J Obstet Gynecol Reprod Biol 2009;147(1):21-4.
Tripp DA, Curtis Nickel J, Katz L. A feasibility trial of a cognitive-behavioural symptom management program for chronic pelvic pain for men with refractory chronic prostatitis/chronic pelvic pain syndrome. Can Urol Assoc J 2011;5(5):328-32.
Fry RP, Crisp AH, Beard RW. Sociopsychological factors in chronic pelvic pain: a review. J Psychosom Res 1997;42(1):1-15.
Franco JVA, Turk T, Jung JH. Non-pharmacological interventions for treating chronic prostatitis/chronic pelvic pain syndrome: a Cochrane systematic review. BJU Int 2018;doi:10.1111/bju.14492 [Epub ahead of print].
Anderson R, Wise D, Sawyer T, et al. Safety and effectiveness of an internal pelvic myofascial trigger point wand for urologic chronic pelvic pain syndrome. Clin J Pain 2011;27(9):764-8.
Van Alstyne LS, Harrington KL, Haskvitz EM. Physical therapist management of chronic prostatitis/chronic pelvic pain syndrome. Phys Ther 2010;90(12):1795-806.
Schaffer SD, Yucha CB. Relaxation & pain management: the relaxation response can play a role in managing chronic and acute pain. AJN 2004;104(8):75-82.
Hofmann SG, Sawyer AT, Witt AA, et al. The effect of mindfulness-based therapy on anxiety and depression: a meta-analytic review. J Consult Clin Psychol 2010;78(2):169-83.
Sutar R, Yadav S, Desai G. Yoga intervention and functional pain syndromes: a selective review. Int Rev Psychiatry 2016;28(3):316-22.
Chiesa A, Serretti A. Mindfulness-based stress reduction for stress management in healthy people: a review and meta-analysis. The Journal of Alternative and Complementary Medicine 2009;15(5):593-600.
Additional sources:
Hard Flaccid Syndrome: Penetrating What We Know ? Entropy Physiotherapy
http://www.corebodyclinic.co.uk/mens...h/hard-flaccid
https://www.harbornephysio.co.uk/bir...=detail&bId=47
https://www.urologynews.uk.com/featu...accid-syndrome
https://chartable.com/podcasts/in-yo...-gerard-greene

zero0x · 05-06-2020, 12:07 AM

I find adductor stretches missing in compilation.
Few users on reddit has a feeling of discomfort in anus area, like going to dump feeling or like a ball stuck there.

Pegasus · 05-06-2020, 04:40 AM

So add abductor stretch.

HansTwilight · 05-29-2020, 06:37 PM

Added to sticky*

Pegasus · 06-18-2020, 09:44 PM

Originally posted by HansTwilight View Post

Added to sticky*

Hans is a leading exponent of self help for the pelvic floor onsite . For those doing self therapy for the pelvic floor his work is recommended .

Pegasus · 08-08-2020, 05:57 AM

https://www.ncbi.nlm.nih.gov/corehtm...bclinandro.png

Basic Clin Androl. 2020; 30: 7.
Published online 2020 Jun 4. doi: 10.1186/s12610-020-00105-5

PMCID: PMC7271516
PMID: 32518654

Language: English | French

Hard flaccid syndrome: state of current knowledge

Maher Abdessater,https://www.ncbi.nlm.nih.gov/corehtm...s/corrauth.gif1 Anthony Kanbar,2 William Akakpo,1 and Sebastien Beley2

Author information Article notes Copyright and License information Disclaimer

Associated Data

Data Availability Statement

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Abstract

Introduction

Hard-flaccid syndrome is gaining increased interest among male sexual dysfunctions in the last years. It is poorly understood and defined. Most of the information comes from online forums. This paper is a review of current knowledge on the clinical presentation, diagnosis, pathophysiological mechanisms and treatments of this newly recognized condition.

Material and methods

A literature review was conducted on MEDLINE, CENTRAL, PASCAL databases and google scholar, using the terms: hard, flaccid, syndrome. The research identified 16 articles published between 2018 and February 2019. After reference lists review and duplicates removal, 7 full text references were eligible and useful for our review that follows PRISMA guidelines.

Results

The condition is acquired, chronic and painful. It is characterized by a constantly semi-rigid penis at the flaccid state and a loss in erectile rigidity. Patients have penile sensory changes, urinary symptoms, erectile dysfunction, pelvic floor muscles contraction and psychological distress. Symptoms are worse in standing position. The majority of the cases aged between their second and third decades. A traumatic injury at the base of an erect penis is the initial event. Neurovascular structures damage and subsequent sensory, muscular and vascular changes follow. Initial symptoms trigger emotional distress and reactional sympathetic stimulation that worsen symptoms. Diagnosis is based on patient’s history. Imaging and blood tests are normal. Differential diagnosis includes high-flow priapism and non-erecting erections. A multimodal treatment has been so far the most beneficial strategy, consisting of behavioral modifications to reduce stress and decrease pelvic floor muscles contraction, evaluation and treatment of the associated psychological conditions, and medical therapy for pain control and the treatment of the associated erectile dysfunction.

Conclusion

Hard-flaccid syndrome is poorly recognized in the daily clinical experience and not well defined. A multimodal approach seems so far the most efficient strategy for treatment. Additional evidence based studies with better quality are needed to define the exact pathophysiological mechanisms and subsequently more efficient therapeutic strategies.

Keywords: Male sexual dysfunction, Hard flaccid, Semi-rigid

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Introduction

Male sexual dysfunctions (MSD) affect man’s sexual and social life and the wellbeing of couples [1]. They are not limited to erectile dysfunction (ED) and premature ejaculations. A new condition called hard-flaccid syndrome (HFS) is gaining increased interest in the last years and being the subject of many online forums. It is a chronic painful condition characterized by a semi-rigid penis at the flaccid state and a loss in erectile rigidity. Patients have penile sensory changes, erectile dysfunction, pelvic floor muscles contraction and psychological distress [1, 2]. HFS remains poorly understood, with no evidence based definition [3].
This paper is a review of current knowledge on the clinical presentation, diagnosis, pathophysiological mechanisms and treatments of this newly recognized condition.

Go to:

Material and methods

A literature review was conducted on MEDLINE, CENTRAL, PASCAL databases and google scholar, using the terms: hard, flaccid, syndrome. The review followed PRISMA guidelines. Neither language, date nor modality restrictions were applied.
The research identified 16 articles published between 2018 and February 2019. Reference lists of relevant articles were also reviewed for additional 1 article. After duplicates removal, 12 titles and abstracts were evaluated, from which 5 were excluded for having irrelevant subjects. Seven full text references were eligible and useful for our review, consisting of: 1 qualitative study utilizing thematic analysis of online forums and blogs, 1 comment on the previous article, 2 case series and 3 online web page articles. In total 6 cases were reported in the 2 case series and 152 online discussions were included in the thematic analysis.
The privacy of users was respected by Gul et al., who included only public forum sites consisting of anonymous users, and excluded the private discussions [4].
Quality of information provided by the websites was evaluated using the Health on the Net code (HONcode), the Journal of the American Medical Association (JAMA) benchmark criteria, and the DISCERN score, and results are detailed in Table 1. The HONcode certification was assessed as present or absent for each website. The JAMA benchmark criteria assessing website authorship, attribution, disclosure, and currency was rated on a scale between 0 and 4 points. The DISCERN score, that is based on 16 questions evaluating publication quality and reliability, varied between 0 and 80 points [5].
Table 1

Evaluation of the quality of the websites included

HON code	Seal	No	No	No
JAMA benchmarks	Authorship	1	1	1
Attribution	1	1	1
Currencies	1	0	1
Disclosure	1	0	1
Total	4	2	4
DISCERN instrument	Are the aims clear?	5	3	5
Does it achieve its aims?	5	3	5
Is it relevant?	5	5	5
Is it clear what sources of information were used to compile the publication (other than the author or producer)?	5	5	5
Is it clear when the information used or reported in the publication was produced?	5	5	5
Is it balanced and unbiased?	5	3	3
Does it provide details of additional sources of support and information?	1	5	1
Does it refer to areas of uncertainty?	5	5	1
Does it describe how each treatment works?	5	3	5
Does it describe the benefits of each treatment?	5	3	5
Does it describe the risks of each treatment?	1	1	1
Does it describe what would happen if no treatment is used?	1	1	1
Does it describe how the treatment choices affect overall quality of life?	5	1	3
Is it clear that there may be more than one possible treatment choice?	5	1	1
Does it provide support for shared decision-making?	1	1	1
Based on the answers to all of the above questions, rate the overall quality of the publication as a source of information about treatment choices	4	1	2
Total	63	46	49

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HON Health on the Net; JAMA Journal of the American Medical Association

Results are presented in a descriptive manner. A flow diagram of the literature review process is presented in Fig. 1. Details and quality of the references included are presented in Table 2.

https://www.ncbi.nlm.nih.gov/pmc/art..._Fig1_HTML.jpg

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Fig. 1
Flow diagram of the literature review process on hard flaccid syndrome

Table 2

Description of the details and the quality of the references included

Gul, 2019 [4]	Qualitative study utilizing thematic analysis of 152 online discussions from internet forums	Data limited to English literature Selection bias of internet users	Symptoms Initial event Management strategies Physiopathology diagram
Yachia, 2019 [6]	Comment on article		Differential diagnosis Relation to chronic pelvis pain syndrome
Gul, 2019 [1]	4 case series		Symptoms Pathophysiological mechanisms Treatment
Gul, 2019 [3]	2 case series		Symptoms
Hughes, 2019 [7]	Academic web page: Urology news	HONcode seal: negative JAMA benchmarks: 4 DISCERN score: 63	Clinical signs and symptoms Management
Bond, 2019 [8]	Commercial web page: Pegasus Gym Blog	HONcode seal: negative JAMA benchmarks: 2 DISCERN score: 46	Clinical presentation Physiopathology diagram
Harville, 2018 [2]	Health professional web page: Entropy Blog	HONcode seal: negative JAMA benchmarks: 4 DISCERN score: 49	Clinical symptoms Pathophysiological mechanisms Management

Open in a separate window

HON Health on the Net; JAMA Journal of the American Medical Association

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Results

Clinical presentation

HFS is an acquired condition characterized by a constantly semi rigid penis at flaccid state. Patients report most commonly odd sensory changes in the penis best described by numbness or coldness with a decreased sensitivity, particularly at the level of the glans [1, 4]. Associated chief complaint is a new onset ED with decreased frequency of morning and nocturnal erections. Excessive visual and physical stimulation are needed to achieve erection, which are difficult to maintain. Typically, a reduction in rigidity is noted on erection, with soft and cold glans. Penile and perineal pain during micturition and ejaculation are reported, and in majority of patients, are worse in the standing position [4]. Patients suffer from emotional distress manifesting by anxiety, depression, decreased libido and insomnia, as it is the case with all the MSDs [4, 7]. Although urinary symptoms are uncommon, a reduction in the urinary flow have been reported [4]. Cases reported in literature age from their late teens to the seventh decade, but the majority are in their second or third decades [7]. Incidence is not well known, since most of the information on HFS is obtained from patients discussing their symptoms on online forums or private chats groups [4].

Pathophysiological mechanisms

Pathophysiological mechanisms are not well defined. Most patients report that their symptoms started after a traumatic event (use of vacuum, tough masturbation or sex, jelqing, excessive squatting), with a varying delay from minutes to weeks. Several patients reported that the traumatic event happened while they were under the effect of drugs like marijuana and bremelanotide [1, 3].
A traumatic injury at the base of an erect penis, of the neurovascular structures, that supply the muscles of the pelvic floor and the penis, was suggested to be the initiating event [6, 7]. The injury of the dorsal artery of the penis, the bulbourethral and the pudendal arteries, and the pudendal and dorsal nerve of the penis cause vascular and sensory changes that are responsible of the partial engorgement of the penis during erections and the odd penile sensations described by the patients [6].
Initial symptoms trigger emotional distress and reactional sympathetic stimulation. The latter is responsible of prolonged pelvic floor muscles spasm that applies additional extrinsic compression of the neurovascular structures with consequent penile hypoxia, neuropraxia, additional sensory changes and impairment of the pelvic muscles [1, 7].
The altered motor function and sustained contraction of the ischiocavernous, bulbomembranous and the external urethral sphincter muscles contribute to the venous outflow obstruction of the penis that is responsible of the semi-hardness state of the flaccid penis. A secondary myoneuropathy with a loss of relaxation ability follows and consequent erectile and ejaculatory dysfunctions develop [1, 7].
Symptoms result in psychological disturbances that affect the libido, erectile function, and general well-being [1]. Figure 2 describes suggests a pathophysiological scenario potentially involving most of the proposed mechanisms in literature.

https://www.ncbi.nlm.nih.gov/pmc/art..._Fig2_HTML.jpg

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Fig. 2
Diagram showing the pathophysiological mechanisms behind the hard flaccid syndrome

The variety of the symptoms and their intensity are related to the location of the lesion and the extension of the inflammation of the injured nerves and vessels in the radix of the penis [1, 4].

Diagnosis

The diagnosis is made based on patient’s history. Physical exam is unremarkable. Engorgement of the penis is revealed on examination. On erection, the glans might remain flaccid [7]. Hardness at the level of penis base is described by some [1, 4]. Imaging, doppler studies and blood tests, including hormonal studies, are normal [4].

Differential diagnoses

Differential diagnoses include high-flow priapism (HFP) and non-erection erections. HFP is caused by arteriovenous fistulas in the penile vasculature, and is seen after blunt or penetrating trauma to the penis. Patients experience partial painless erections like those seen in HFS, but in HFP the doppler ultrasound can localize the rupture and the arteriovenous fistula [1]. Non-erecting erections were described by Yachia in 2009. They are caused by the deficiency or the absence of the suspensory ligament of the penis. Clinically this condition manifests by the lack of elevation of penis on erection. It is usually congenital or less frequently acquired after a trauma, and it is repaired surgically by corporopexy [6].

Treatment

The treatment of this conditions is not well defined yet. Multiple therapeutic modalities were suggested, but were not equally efficient in all patients [4]. A multimodal treatment have so far been the most beneficial strategy [7].
The evaluation and treatment of the associated psychological conditions is crucial because stress and anxiety trigger additional sympathetic stimulation, and symptoms deterioration have been reported in periods of elevated stress [7]. Behavioral modifications (good sleep, healthy eating, regular exercises), biofeedback, cognitive behavioral therapies, breathing exercises, yoga reduce stress, improve well-being and decrease pelvic floor muscles contraction [7]. Pain can be controlled by analgesics. Medications like phosphodiesterase 5 inhibitors and antidepressants help treating associated erectile dysfunction and psychological conditions. Low-intensity shock wave therapy [1] has been temporarily alleviating in some patients who failed other therapies.

Limitations

Our review was limited only to English and French references, and most of the analysis was obtained from internet forums. Although the collection of data from online patient’s forum is considered valid when literature is not available [9] and very useful for the description of the clinical aspects of a phenomenon [4], it has several limitations. First of all, selection bias is inevitable since only internet users are included. Another major drawback is deindividuation, because users tend to have more extreme and offensive tone on online blogs than they would in real life [9]. The online forum analysis lacks socio-demographic information which are of high importance when reviewing a medical pathology. Furthermore, the informed consent was not obtained from users who may be annoyed by the analysis of their posts. Overall, the quality of the websites included was not equal and not exclusively high. It was assessed using the HON code, the JAMA benchmark criteria, and the DISCERN score. None had a HON code certification, and the JAMA benchmarks and the DISCERN score varied between the three websites as detailed in Table Table11.

Go to:

Conclusion

HFS has been the subject of discussion and debate on many male forums [8]. It is not recognized by sexual medicine community [1], and poorly recognized in the daily clinical experience [4]. Many patients are suffering from this conditions, and the majority is misdiagnosed and left untreated [7]. An initial traumatic event with the resulting inflammatory response is thought to generate stress that is a key factor is developing a prolonged contraction of the pelvic muscles. It is difficult to treat. A multimodal approach seems so far the most efficient strategy [8]. Physical therapy seems to play important role in the relaxation of the pelvic floor muscles [2]. Additional evidence based studies are needed to define the exact pathophysiological mechanisms and subsequently give more efficient therapeutic strategies.

Go to:

Acknowledgements

Not applicable.

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Abbreviations

ED	Erectile dysfunction
HFP	High-flow priapism
HFS	Hard flaccid syndrome
HON	Health on the Net
JAMA	Journal of the American Medical Association
MSD	Male sexual dysfunction

Pegasus · 08-08-2020, 07:07 PM

From a physio in New York.
Quote
physical or psychological stressor can trigger an emotional response, which can then stimulate the pelvic muscles and cause a sustained muscle contraction. This can then lead to a shortening of the contractile tissues of the penis, thus leading to tightness of the surrounding muscles and tissue and then cause pain. A traumatic event, such as rough masturbation, can lead to injury of nerves and pelvic floor musculature, which can contribute to the onset of symptoms. This can then lead to psychological problems which can result in erectile dysfunction and decreased libido.
Stress and anxiety about the condition can also exacerbate the condition. Chronic pain can change behaviors into catastrophic thinking about the condition. This can cause more muscle tension and and increased feelings of helplessness and negativity. Patients with chronic pelvic pain are likely to remain catastrophic in their thinking about pain for prolonged periods if they don’t or are unable to seek help.
Pelvic floor physical therapy can help men with hard flaccid. Treatment sessions can include internal and external pelvic floor trigger point release, learning relaxation strategies to reduce stress, and gentle core strengthening exercises to decrease stress on the pelvic floor musculature.

Written by: Roseanne Cruz Schoen, PT, DPT
References:

Pegasus · 08-08-2020, 07:11 PM

Notice the continued mention of head issues in HF , post 10 uses the phrase "catastrophic thinking". I see a lot of this onsite and it really does prevent effective treatment .

Butters65 · 08-11-2020, 12:53 PM

Thanks for posting this Pegasus - will definitely take time and read through it later tonight -

Trazohell · 08-17-2020, 09:24 AM

Totally bullshit. Yoga, relaxing, stretching, no fap, massages, therawand, diet (lol) will never cure a real hard flaccid. I did them all together and they did literally nothing. Only DCT did a bit but's it seems not like a cure also.

Ultimate Burrito · 08-17-2020, 09:29 AM

Originally posted by Trazohell View Post

Totally bullshit. Yoga, relaxing, stretching, no fap, massages, therawand, diet (lol) will never cure a real hard flaccid. I did them all together and they did literally nothing. Only DCT did a bit but's it seems not like a cure also.

Please review the PEGYM code and conduct here: https://www.pegym.com/forums/beginne...cess-here.html

It's okay to disagree but be respectful.

closed297 · 08-17-2020, 09:47 AM

Totally bullshit. Yoga, relaxing, stretching, no fap, massages, therawand, diet (lol) will never cure a real hard flaccid. I did them all together and they did literally nothing.

I'd say even if it isn't a cure to your issue it has a lot of OTHER benefits..

Saying it does NOTHING is pretty close minded..

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